Colitis Thrombophlebitis

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Colitis Thrombophlebitis What is Phlebitis? Treatment & Symptoms for Thrombophlebitis

Deep vein thrombosis DVT definition and facts, Colitis Thrombophlebitis. It is important to know the body's anatomy and function Colitis Thrombophlebitis understand why clots form in veins and why they can be dangerous.

What does a blood clot in the leg look like? Blood is meant to flow. If it becomes stagnant, there is a potential for it to clot. The blood in veins constantly forms microscopic clots that are routinely broken down by the body. If the balance of clot formation and clot breakdown is altered, significant clotting may occur. A thrombus can form if one or a combination of the following situations. Leg swelling generally occurs because of an abnormal Colitis Thrombophlebitis of fluid in the tissues of the lower extremity, Colitis Thrombophlebitis.

Signs and symptoms of DVT. The signs and symptoms of DVT are related to obstruction of blood returning to the heart and causing a backup of blood in the leg. You Colitis Thrombophlebitis or may not have all of these symptoms, or your may have none, Colitis Thrombophlebitis.

The symptoms of the condition may mimic an infection or cellulitis of the arm or leg, Colitis Thrombophlebitis. In the past doctors and other health care professionals perform simple tests on patients to make a diagnosis of a blood clot in the leg; however, they have not been effective. For example, pulling the patient's toes toward the nose Homans' signand squeezing the calf to produce pain Pratt's sign.

Today, doctors and health care professionals usually do not rely upon whether these signs and symptoms are present to make the diagnosis or decide that you have DVT. Signs and symptoms of superficial blood clots. Blood clots in the superficial vein system closer to the surface of the skinmost often occur due to trauma to the vein, which causes a small blood clot to form, Colitis Thrombophlebitis. Inflammation of the vein and surrounding skin causes the symptoms similar to any other type of inflammation, Colitis Thrombophlebitis, for example.

You often can feel the vein as a firm, thickened cord. There may be inflammation that follows the course of part of the leg vein, Colitis Thrombophlebitis. Although there is inflammation, there is no infection. Varicosities can predispose to superficial thrombophlebitis and varicose veins. This occurs when the valves of the larger veins in Colitis Thrombophlebitis superficial system fail the greater and lesser saphenous veinswhich allows blood to back up and cause the veins to swell and become distorted or tortuous.

The valves fail when veins lose their elasticity and stretch. This can Colitis Thrombophlebitis due to age, prolonged standing, obesity, Colitis Thrombophlebitis, pregnancy, and Colitis Thrombophlebitis factors.

Who is at risk? There are a wide variety of people who are at risk for developing blood clots. Some risk factors include:. Which types of doctors treat DVT? People with a swollen extremity or concern that a DVT exists may be cared for by a variety of health-care professionals. Both the primary care provider including internal medicine and family medicine specialists and a health care professional at in an urgent care walk in clinic or emergency department are able to recognize and diagnose the condition.

Some people go to the hospital and the diagnosis is made there. Treatment is usually started by the doctor who makes the diagnosis, but long-term treatment decisions, Colitis Thrombophlebitis, Colitis Thrombophlebitis stratification, Colitis Thrombophlebitis, and follow-up usually is be done by the person's primary care doctor.

Depending upon the situation, Colitis Thrombophlebitis, a hematologist specialist in blood disorders may be consulted. If there is need for the clot to be removed or dissolved, an interventional radiologist may also be involved. Depending upon the medication used to anticoagulate the blood, Colitis Thrombophlebitis, pharmacists and anticoagulation nurses may also be involved on your treatment team.

What tests diagnose the condition? The diagnosis of superficial thrombophlebitis usually is made by the doctor at the bedside of the patient, based upon history, Colitis Thrombophlebitis, potential risk factors present, and findings from the physical examination. Further risk stratification tools may include scoring systems that can help decide whether a DVT is likely. D-dimer is a blood test that may be used as a screening test to Colitis Thrombophlebitis if a blood clot exists.

D-dimer is a chemical that is produced when a blood clot in the body gradually dissolves, Colitis Thrombophlebitis. The test is used as a positive or negative indicator. If the result is negative, then in most cases no blood clot exists. If the Colitis Thrombophlebitis test is positive, it does not necessarily mean that a deep vein thrombosis is present since many situations will have an expected positive result. Any bruise or blood clot will result in a positive D-dimer result for example, from surgery, a fall, in cancer or in pregnancy, Colitis Thrombophlebitis.

For that reason, D-dimer testing must be used selectively. What are the Colitis Thrombophlebitis guidelines for DVT? The treatment for deep venous thrombosis is anticoagulation or "thinning the blood" with medications, Colitis Thrombophlebitis. The recommended length of treatment for an uncomplicated DVT is three months.

Depending upon the patient's situation, underlying medical conditions and the reason for developing a blood clot, a longer duration of anticoagulation may be required. At three months, the doctor or other health care professional should evaluate the patient in regard to the potential for future blood clot formation. There are times when anticoagulation may have increased bleeding risk, for example, if the patient has had recent major surgery anticoagulation thins all of the blood in the body Colitis Thrombophlebitis just the DVT.

Other Colitis Thrombophlebitis risks occur in Colitis Thrombophlebitis with liver disease and those who take medications that can interact with the anticoagulation medicines.

What is the treatment of superficial blood clots? If the thrombophlebitis occurs near the groin where the superficial and deep systems join together, there is potential that the thrombus could extend into the deep venous system.

These patients may require anticoagulation or blood thinning therapy. Medications to treat blood clots Colitis Thrombophlebitis the leg. Anticoagulation prevents further growth of the blood clot and prevents it from forming an embolus that can travel to the lung, Colitis Thrombophlebitis. The body has a complex mechanism to form blood clots to help repair blood vessel damage, Colitis Thrombophlebitis. There is a clotting cascade with numerous blood factors that Colitis Thrombophlebitis to be activated for a clot to form.

There are difference types of medications that can be used for anticoagulation to treat DVT:. The American College of Chest Physicians has guidelines that give direction as to what medications might best be used in different situations.

If active cancer exists, the treatment of DVT would be with enoxaparin as the drug of first choice. NOACs work almost immediately to thin the blood and anticoagulate the patient. There is no need for blood tests to monitor dosing. The NOAC medications presently approved for deep vein thrombosis treatment include:.

All four are also indicated to treat pulmonary embolism. They also may be prescribed to patients anticoagulated with nonvalvular atrial fibrillation to prevent stroke and systemic embolus. Historically, it was a first-line medication for treating blood clots, Colitis Thrombophlebitis, but its Was ist gefährlich uterine Krampfadern has been diminished because of the availability of newer drugs.

While warfarin may be prescribed immediately after the diagnosis of DVT, it takes up to Colitis Thrombophlebitis week or more for it to reach therapeutic levels in the blood so that the blood is appropriately thinned.

Therefore, Colitis Thrombophlebitis, low Colitis Thrombophlebitis weight heparin enoxaparin [Lovenox ] is administered at Colitis Thrombophlebitis same time. Enoxaparin thins the blood almost immediately and is used as a bridge Colitis Thrombophlebitis until the warfarin has taken effect. Enoxaparin injections can be given Colitis Thrombophlebitis an outpatient basis.

For those patients who have contraindications to the use of enoxaparin for example, kidney failure does not allow the Colitis Thrombophlebitis to be appropriately metabolizedintravenous heparin can be used as the first step in association with warfarin. This requires admission to the hospital. The dosage of warfarin is monitored by blood tests measuring the prothrombin time PTor INR international normalized ratio. Side effects and risks of anticoagulation therapy.

Patients who take anticoagulation medications are at risk for bleeding. The decision to use these medications must balance the Colitis Thrombophlebitis and rewards of the treatment. Should bleeding occur, there are strategies available to reverse the anticoagulation effects. Some patients may have contraindications to anticoagulation therapy, for example a patient with bleeding in the brain, major trauma, or recent significant surgery.

An alternative may be to place a filter in the inferior vena cava the major vein that collects blood from both legs to prevent emboli, should they arise, from reaching the heart and lungs.

These filters may be effective but have the potential risk of being the source of new clot formation. Does DVT require surgery? Surgery is a rare option in treating large deep venous thrombosis of the leg Colitis Thrombophlebitis patients who cannot take blood thinners or who have developed recurrent blood clots while on anti-coagulant medications.

The surgery is usually accompanied by placing an IVC inferior vena cava filter to prevent future clots from Colitis Thrombophlebitis to the lung.

Phlegmasia Cerulea Dolens describes a situation in which a blood clot forms in the iliac vein of the pelvis and the femoral vein of the leg, obstructing almost all Colitis Thrombophlebitis return and compromising Colitis Thrombophlebitis supply to the leg. In this case surgery may be considered to remove the clot, but the patient will also require anti-coagulant medications. Stents may also be required to keep a vein open and prevent clotting.

May Thurner Syndrome, also known as iliac vein compression syndrome, Colitis Thrombophlebitis, is a cause of phlegmasia, in which the iliac vein in the pelvis is compressed and a stent is also needed.

What are the complications? Pulmonary embolism is the major complication of deep vein thrombosis. With signs and symptoms such as chest pain and shortness of breathColitis Thrombophlebitis, it is a life-threatening condition. Most often pulmonary emboli arise from the legs. Post-phlebitic syndrome can occur after a deep vein thrombosis.

The affected leg or arm can become chronically swollen and painful with skin color changes and ulcer formation around the foot and ankle, Colitis Thrombophlebitis. How can blood clots in the legs be prevented? Harrison's Principles of Internal Medicine, 19th Ed.


Swelling is an increase in the size or a change in the shape of an area of the body. Swelling can be caused by collection of body fluid, tissue growth, or abnormal.

See related handout on skin and soft tissue infectionswritten by the authors of this article, Colitis Thrombophlebitis. Skin and soft tissue infections result from microbial invasion of the skin and its supporting structures. Management is determined by the severity and location of the infection and by patient comorbidities. Infections can be classified as simple uncomplicated or complicated necrotizing or nonnecrotizingor as suppurative or nonsuppurative.

Most community-acquired infections are caused by Colitis Thrombophlebitis Staphylococcus aureus and beta-hemolytic streptococcus. Simple infections are usually monomicrobial and present with localized clinical findings.

In contrast, complicated infections can be mono- or polymicrobial and may present with systemic inflammatory response syndrome. The diagnosis is based on clinical evaluation. Laboratory testing may be required to confirm an uncertain diagnosis, Colitis Thrombophlebitis, evaluate for deep infections or sepsis, determine the need for inpatient care, and evaluate and treat comorbidities.

Initial antimicrobial choice is empiric, and in simple infections should cover Staphylococcus and Streptococcus species, Colitis Thrombophlebitis. Patients with complicated infections, including suspected necrotizing fasciitis and gangrene, require empiric polymicrobial antibiotic coverage, inpatient treatment, and surgical consultation for debridement.

Superficial and small abscesses respond well to drainage and seldom require antibiotics. Immunocompromised patients require early treatment and antimicrobial coverage for possible atypical organisms. Skin and soft tissue infections SSTIs account for more than 14 million physician office visits each year in the United States, as well as emergency department visits and hospitalizations.

Blood cultures seldom change treatment and are not required in healthy immunocompetent patients with SSTIs. Uncomplicated purulent SSTIs in easily Colitis Thrombophlebitis areas without overlying cellulitis can be treated with incision and drainage only; antibiotic therapy does not improve outcomes.

Inpatient treatment is recommended for patients with uncontrolled SSTIs despite adequate oral antibiotic therapy; those who cannot tolerate oral antibiotics; those who require surgery; those with initial severe or complicated SSTIs; and those with underlying unstable comorbid illnesses or signs of systemic sepsis.

Treatment of necrotizing fasciitis involves early recognition and surgical debridement of necrotic tissue, combined with high-dose broad-spectrum intravenous antibiotics. For information about the SORT evidence rating system, go to http: Avoid antibiotics and wound cultures in emergency department patients with uncomplicated skin and soft tissue abscesses after successful incision Colitis Thrombophlebitis drainage and with adequate medical follow-up.

For more information on the Choosing Wisely Campaign, see http: For supporting citations and to search Choosing Wisely recommendations relevant to primary care, see http: SSTIs are classified as simple uncomplicated or complicated necrotizing or nonnecrotizing and can involve the skin, subcutaneous fat, fascial layers, and musculotendinous structures. Information from reference 3.

Simple infections confined to the skin and underlying superficial soft tissues generally respond well to outpatient management. Common simple SSTIs include cellulitis, Colitis Thrombophlebitis, impetigo, ecthyma, folliculitis, furuncles, carbuncles, abscesses, and trauma-related infections 6 Figures 1 through 3.

Complicated infections extending into and involving the underlying deep Colitis Thrombophlebitis include deep abscesses, decubitus ulcers, necrotizing fasciitis, Fournier gangrene, and infections from human or animal bites 7 Figure 4. These infections may present with features of systemic inflammatory response syndrome or sepsis, Colitis Thrombophlebitis, occasionally, ischemic necrosis.

Perianal infections, diabetic foot infections, infections in patients with significant comorbidities, and infections from resistant pathogens also represent complicated infections. Older age, Colitis Thrombophlebitis, cardiopulmonary or hepatorenal disease, diabetes mellitus, debility, immunosenescence or immunocompromise, obesity, peripheral arteriovenous or lymphatic insufficiency, and trauma are among the risk factors for SSTIs Table 2.

Information from references 9 through Predisposing factors for SSTIs include Colitis Thrombophlebitis tissue vascularity and oxygenation, increased peripheral fluid stasis and risk of skin trauma, and decreased ability to combat infections. For example, diabetes increases the risk of infection-associated complications fivefold, Colitis Thrombophlebitis.

Staphylococcus aureusStreptococcusanaerobes often polymicrobial. Polymicrobial Bacteroides, Bartonella henselae, Capnocytophaga canimorsus, Eikenella corrodens, Pasteurella multocida, Peptostreptococcus, S. Traumatic or spontaneous; severe pain at injury site followed by skin changes e. Beta-hemolytic streptococci, Haemophilus influenzae childrenS. Candida, dermatophytes, Colitis Thrombophlebitis, Pseudomonas aeruginosaColitis Thrombophlebitis, S.

Infection or inflammation of the hair follicles; tends to occur in areas with increased sweating; associated with acne Colitis Thrombophlebitis steroid use; painful or painless pustule with underlying swelling. Walled-off collection of pus; painful, firm swelling; systemic features of infection; carbuncles are larger, deeper, and involve skin and subcutaneous tissue over thicker skin of neck, back, and lateral thighs, and drain through multiple pores.

Common in infants and children; affects skin of nose, Colitis Thrombophlebitis, mouth, or limbs; mild soreness, redness, vesicles, and crusting; may cause glomerulonephritis; vesicles may enlarge bullae ; may spread to lymph nodes, bone, joints, or lung.

Mental status changes and hypotension suggest worsening sepsis and hemodynamic compromise. Information from Colitis Thrombophlebitis 5 and Most SSTIs occur de novo, or follow a breach in the protective skin barrier from trauma, surgery, or increased tissue tension secondary to fluid stasis.

The infection may also originate from an adjacent site or from embolic spread from a distant site. In one prospective study, Colitis Thrombophlebitis, beta-hemolytic streptococcus was found to cause nearly three-fourths of cases of diffuse cellulitis.

Lymphatic and hematogenous dissemination causes septicemia and spread to other organs e. Diabetic lower limb infections, severe hospital-acquired infections, necrotizing infections, Colitis Thrombophlebitis, and head and hand infections pose Colitis Thrombophlebitis risks of mortality and functional disability.

Patients with simple SSTIs present with erythema, warmth, edema, Colitis Thrombophlebitis, and pain over the affected site. Systemic features of infection may follow, their intensity reflecting the magnitude of infection.

The lower extremities are most commonly involved. Patients with necrotizing fasciitis may have pain disproportionate to the physical findings, rapid progression of infection, cutaneous anesthesia, Colitis Thrombophlebitis or bullous changes, and crepitus indicating gas in the soft tissues. The diagnosis of SSTIs is predominantly clinical.

A complete blood count, C-reactive protein level, Colitis Thrombophlebitis, and liver and kidney function Colitis Thrombophlebitis should be ordered for patients with severe infections, and for those with comorbidities causing organ dysfunction. Maximum score is Scores of 6 or more are indicative of necrotizing fasciitis, and scores of 8 or more are highly predictive.

Blood cultures are unlikely to change the management of simple localized SSTIs in otherwise healthy, immunocompetent patients, and are typically unnecessary. Sterile aspiration of infected tissue is another recommended sampling method, preferably before commencing antibiotic therapy. Imaging studies are not indicated for simple SSTIs, and surgery should not be delayed for imaging.

Plain radiography, Colitis Thrombophlebitis, ultrasonography, computed tomography, or magnetic resonance imaging may show soft tissue edema or fascial thickening, fluid collections, or soft tissue air. The management of SSTIs is determined primarily by their severity and location, and by the patient's comorbidities Figure 5.

According to guidelines from the Infectious Diseases Society of America, initial management is determined by the presence or absence of purulence, acuity, and type of infection. Initial management of skin and soft tissue infections, Colitis Thrombophlebitis. Children younger than 3 months and less than 40 kg 89 lb: For MSSA infections and human or animal bites. For MSSA infections, impetigo, and human or animal bites; twice-daily dosing is an option.

Doxycycline or minocycline Minocin. For MRSA infections and human or animal bites; not recommended for children younger than 8 years. Clostridium difficile colitis, hepatotoxicity, pseudotumor cerebri, Stevens-Johnson syndrome. For human or animal bites; not useful in MRSA infections; not recommended for children, Colitis Thrombophlebitis. For MRSA impetigo and folliculitis; not recommended for children younger than 2 months.

For MSSA impetigo; not recommended for children younger than 9 months. For MRSA infections and human or animal bites; contraindicated in children younger than 2 months, Colitis Thrombophlebitis. Mild purulent SSTIs in easily accessible areas without significant overlying cellulitis can be treated with incision and drainage alone. Antibiotic therapy is required Ich bin 16 I Krampfadern abscesses that are associated with extensive cellulitis, rapid progression, or poor response to initial drainage; that involve specific sites e.

Inpatient treatment is necessary for patients who have uncontrolled infection despite adequate outpatient antimicrobial therapy or who cannot tolerate oral antibiotics Figure 6. Hospitalization is also indicated for patients who initially present with severe or complicated infections, unstable comorbid illnesses, or signs of systemic sepsis, or who need surgical intervention under anesthesia.

Intravenous antibiotics should be continued until the clinical picture improves, the patient can tolerate oral intake, and drainage or debridement is completed. The recommended duration of antibiotic therapy for hospitalized patients is seven to 14 days, Colitis Thrombophlebitis.

Inpatient management of skin and soft tissue infections. Used with metronidazole Flagyl or clindamycin for initial treatment of polymicrobial necrotizing infections, Colitis Thrombophlebitis. Dose adjustment required in patients with renal impairment. Useful in waterborne infections; used with doxycycline for Aeromonas hydrophila and Vibrio vulnificus infections.

Adults and children 12 years and older: Children 8 years and older and less than 45 kg lb: Useful in waterborne infections; used with ciprofloxacin Ciproceftriaxone, or cefotaxime in A. Used with cefotaxime for initial treatment of Colitis Thrombophlebitis necrotizing infections.

For necrotizing Salbe mit trophischen Geschwüren des Fußes caused by sensitive staphylococci. Combined therapy for necrotizing fasciitis caused by streptococci; either drug is effective in clostridial infections, Colitis Thrombophlebitis.

Rare adverse effects of clindamycin: First-line antimicrobial for treating polymicrobial necrotizing infections. For MRSA infections; increases mortality risk; considered medication of last resort.

Parenteral drug of choice for MRSA infections in patients allergic to penicillin; 7- to day course for skin and Colitis Thrombophlebitis tissue infections; 6-week course for bacteremia; maintain trough levels at 10 to 20 mg per L. Treatment of necrotizing fasciitis involves early recognition and surgical consultation for debridement of necrotic tissue combined with empiric high-dose intravenous broad-spectrum antibiotics, Colitis Thrombophlebitis. Monomicrobial necrotizing fasciitis caused by streptococcal and clostridial infections is treated with penicillin G and clindamycin; S.

Antibiotic therapy should be continued until features of sepsis have resolved and surgery is completed. Patients may require repeated surgery until debridement and drainage are complete and healing has commenced.

Immunocompromised patients are more prone to SSTIs and may not demonstrate classic clinical features and laboratory findings because of their attenuated inflammatory response. Diagnostic testing should be performed early to identify the causative organism and evaluate the extent of involvement, and antibiotic therapy should be commenced to cover possible pathogens, including atypical organisms that can cause serious infections e.


Thromboembolic Disease: Deep Vein Thrombosis and Pulmonary Embolism

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